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• W3082015369 abstract "Summary Hydrogen sulphide (H 2 S) is the latest identified small gaseous mediator enabled by its lipophilic nature to freely permeate the biological membranes. Initially, H 2 S was recognized by its roles in neuronal activity and vascular relaxation, which makes it an important molecule involved in paracrine signalling pathways. Recently, the immune regulatory function of gasotransmitters, H 2 S in particular, is increasingly being appreciated. Endogenous H 2 S level has been linked to macrophage activation, polarization and inflammasome formation. Mechanistically, H 2 S‐induced protein S‐sulphydration suppresses several inflammatory pathways including NF‐κB and JNK signalling. Moreover, H 2 S serves as a potent cellular redox regulator to modulate epigenetic alterations and to promote mitochondrial biogenesis in macrophages. Here in this review, we intend to summarize the recent advancements of H 2 S studies in macrophages, and to discuss with focus on the therapeutic potential of H 2 S donors by targeting macrophages. The feasibility of H 2 S signalling component as a macrophage biomarker under disease conditions would be also discussed." @default.
• W3082015369 created "2020-09-08" @default.
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• W3082015369 date "2020-10-06" @default.
• W3082015369 modified "2023-10-17" @default.
• W3082015369 title "The role of hydrogen sulphide signalling in macrophage activation" @default.
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• W3082015369 doi "https://doi.org/10.1111/imm.13253" @default.
• W3082015369 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/7730026" @default.
• W3082015369 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/32876334" @default.
• W3082015369 hasPublicationYear "2020" @default.
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