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- W3082726443 endingPage "5275" @default.
- W3082726443 startingPage "5263" @default.
- W3082726443 abstract "Similar to its predecessors, coronavirus disease 2019 (COVID-19) exhibits neurotrophic properties, which lead to progression of neurologic sequelae. Besides direct viral invasion to the central nervous system (CNS), indirect CNS involvement through viral-mediated immune response is plausible. Aberrant immune pathways such as extreme release of cytokines (cytokine storm), autoimmunity mediated by cross-reactivity between CNS components and viral particles, and microglial activation propagate CNS damage in these patients. Here, we review the currently available evidence to discuss the plausible immunologic pathways that may contribute to the development of COVID-19 neurological complications, namely Alzheimer's disease, Parkinson's disease, stroke, multiple sclerosis, Guillain-Barre syndrome, seizure, and brainstem involvement." @default.
- W3082726443 created "2020-09-08" @default.
- W3082726443 creator A5030768137 @default.
- W3082726443 creator A5076132151 @default.
- W3082726443 creator A5086449868 @default.
- W3082726443 date "2020-09-01" @default.
- W3082726443 modified "2023-10-01" @default.
- W3082726443 title "Understanding the Immunologic Characteristics of Neurologic Manifestations of SARS-CoV-2 and Potential Immunological Mechanisms" @default.
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