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- W3082857741 abstract "<ns4:p>Secondary hyperparathyroidism is a complex pathology that develops as chronic kidney disease progresses. The retention of phosphorus and the reductions in calcium and vitamin D levels stimulate the synthesis and secretion of parathyroid hormone as well as the proliferation rate of parathyroid cells. Parathyroid growth is initially diffuse but it becomes nodular as the disease progresses, making the gland less susceptible to be inhibited. Although the mechanisms underlying the pathophysiology of secondary hyperparathyroidism are well known, new evidence has shed light on unknown aspects of the deregulation of parathyroid function. Secondary hyperparathyroidism is an important feature of chronic kidney disease–mineral and bone disorder and plays an important role in the development of bone disease and vascular calcification. Thus, part of the management of chronic kidney disease relies on maintaining acceptable levels of mineral metabolism parameters in an attempt to slow down or prevent the development of secondary hyperparathyroidism. Here, we will also review the latest evidence regarding several aspects of the clinical and surgical management of secondary hyperparathyroidism.</ns4:p>" @default.
- W3082857741 created "2020-09-08" @default.
- W3082857741 creator A5087558480 @default.
- W3082857741 creator A5090524109 @default.
- W3082857741 date "2020-09-01" @default.
- W3082857741 modified "2023-10-17" @default.
- W3082857741 title "Recent advances in understanding and managing secondary hyperparathyroidism in chronic kidney disease" @default.
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- W3082857741 doi "https://doi.org/10.12688/f1000research.22636.1" @default.
- W3082857741 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/7463297" @default.
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