FRINK Linked Data Fragments server

Matches in SemOpenAlex for { <https://semopenalex.org/work/W3084111603> ?p ?o ?g. }

• W3084111603 abstract "Background:There is growing consensus that pain in pediatric inflammatory bowel disease (IBD) is not fully explained by disease-related processes. However, previous studies have largely measured individual biological, psychological, or social risk factors for pain in isolation. Further, not all youth with IBD presenting to clinic will report presence of pain, and those who do vary in their reports of pain intensity. This study therefore extends prior research by determining biopsychosocial correlates of both presence and intensity of pain in adolescents with IBD, in order to inform targeted pain management intervention approaches. Methods:Adolescents with IBD followed at SickKids, Toronto, and their parents were consecutively enrolled from outpatient clinic. IBD characteristics (diagnosis, time since diagnosis, patient-reported disease activity) were collected. Adolescents reported on current pain (NRS-10), internalizing symptoms (Strengths and Difficulties Questionnaire), and pain catastrophizing (Pain Catastrophizing Scale-Child). Parents reported on protective responses to child pain (Adult Responses to Child Pain) and pain catastrophizing (Pain Catastrophizing Scale-Child). Hurdle models were conducted to examine predictors of presence and intensity of pain in the same model. Biological (patient-reported disease activity, IBD diagnosis subtype, illness duration), psychological (internalizing symptoms, pain catastrophizing) and social (parent pain catastrophizing, parent protective responses) factors were entered as predictors, adjusting for age and sex. Results:Participants included 100 adolescents (12-18; Mean=15 years) with IBD (60% Crohn’s Disease, 40% Ulcerative Colitis or IBD-unclassified) and 76 parents. The majority of the sample was in clinical remission or reported minimal symptoms. Half of participants reported no current pain; for those reporting pain, intensity ranged 1-7 (M=3.43, SD=1.98). Disease activity (OR=53.91,p<.001) and adolescent internalizing symptoms (OR=7.62,p=.03) were significant predictors of presence of pain. Disease activity (RR=1.37,p=.03) and parent protective responses (RR=1.45,p=.02) were significant predictors of intensity of pain. Conclusions:Results suggest that the experience of pain in pediatric IBD is biopsychosocially determined. Patient-reported disease activity and internalizing symptoms predicted presence of pain, while disease activity and parent protective responses predicted intensity of pain. While medical intervention in pediatric IBD is focused on disease management, results suggest that depression/anxiety symptoms as well as parent protective responses may be important targets of pain management interventions in pediatric IBD." @default.
• W3084111603 created "2020-09-14" @default.
• W3084111603 creator A5008230538 @default.
• W3084111603 creator A5020538296 @default.
• W3084111603 creator A5077895731 @default.
• W3084111603 creator A5079379686 @default.
• W3084111603 creator A5079398615 @default.
• W3084111603 date "2020-09-08" @default.
• W3084111603 modified "2023-10-04" @default.
• W3084111603 title "Biopsychosocial Correlates of Presence and Intensity of Pain in Adolescents With Inflammatory Bowel Disease" @default.
• W3084111603 cites W1950114924 @default.
• W3084111603 cites W1995856724 @default.
• W3084111603 cites W2006195533 @default.
• W3084111603 cites W2014023768 @default.
• W3084111603 cites W2018883975 @default.
• W3084111603 cites W2033081617 @default.
• W3084111603 cites W2033582886 @default.
• W3084111603 cites W2041423270 @default.
• W3084111603 cites W2044687532 @default.
• W3084111603 cites W2050648620 @default.
• W3084111603 cites W2058706903 @default.
• W3084111603 cites W2061443531 @default.
• W3084111603 cites W2063756593 @default.
• W3084111603 cites W2066523101 @default.
• W3084111603 cites W2066783177 @default.
• W3084111603 cites W2073167808 @default.
• W3084111603 cites W2073334011 @default.
• W3084111603 cites W2093082052 @default.
• W3084111603 cites W2094138397 @default.
• W3084111603 cites W2108096958 @default.
• W3084111603 cites W2119778450 @default.
• W3084111603 cites W2130155416 @default.
• W3084111603 cites W2138656933 @default.
• W3084111603 cites W2160662805 @default.
• W3084111603 cites W2162433884 @default.
• W3084111603 cites W2276533368 @default.
• W3084111603 cites W2326825446 @default.
• W3084111603 cites W2329480171 @default.
• W3084111603 cites W2335484472 @default.
• W3084111603 cites W2508908484 @default.
• W3084111603 cites W2557644139 @default.
• W3084111603 cites W2590831482 @default.
• W3084111603 cites W2591547055 @default.
• W3084111603 cites W2595976559 @default.
• W3084111603 cites W2753632793 @default.
• W3084111603 cites W2755344236 @default.
• W3084111603 cites W2768878405 @default.
• W3084111603 cites W2786608165 @default.
• W3084111603 cites W2793437768 @default.
• W3084111603 cites W2889059924 @default.
• W3084111603 cites W2919299432 @default.
• W3084111603 cites W2944667314 @default.
• W3084111603 cites W2951763634 @default.
• W3084111603 cites W2982641487 @default.
• W3084111603 cites W2987479881 @default.
• W3084111603 cites W3009260571 @default.
• W3084111603 cites W3028814979 @default.
• W3084111603 doi "https://doi.org/10.3389/fped.2020.00559" @default.
• W3084111603 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/7506075" @default.
• W3084111603 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/33014942" @default.
• W3084111603 hasPublicationYear "2020" @default.
• W3084111603 type Work @default.
• W3084111603 sameAs 3084111603 @default.
• W3084111603 citedByCount "8" @default.
• W3084111603 countsByYear W30841116032021 @default.
• W3084111603 countsByYear W30841116032022 @default.
• W3084111603 countsByYear W30841116032023 @default.
• W3084111603 crossrefType "journal-article" @default.
• W3084111603 hasAuthorship W3084111603A5008230538 @default.
• W3084111603 hasAuthorship W3084111603A5020538296 @default.
• W3084111603 hasAuthorship W3084111603A5077895731 @default.
• W3084111603 hasAuthorship W3084111603A5079379686 @default.
• W3084111603 hasAuthorship W3084111603A5079398615 @default.
• W3084111603 hasBestOaLocation W30841116031 @default.
• W3084111603 hasConcept C112143881 @default.
• W3084111603 hasConcept C118552586 @default.
• W3084111603 hasConcept C126322002 @default.
• W3084111603 hasConcept C137886200 @default.
• W3084111603 hasConcept C1862650 @default.
• W3084111603 hasConcept C2778260677 @default.
• W3084111603 hasConcept C2778271842 @default.
• W3084111603 hasConcept C2779134260 @default.
• W3084111603 hasConcept C2780479503 @default.
• W3084111603 hasConcept C2781118164 @default.
• W3084111603 hasConcept C71924100 @default.
• W3084111603 hasConceptScore W3084111603C112143881 @default.
• W3084111603 hasConceptScore W3084111603C118552586 @default.
• W3084111603 hasConceptScore W3084111603C126322002 @default.
• W3084111603 hasConceptScore W3084111603C137886200 @default.
• W3084111603 hasConceptScore W3084111603C1862650 @default.
• W3084111603 hasConceptScore W3084111603C2778260677 @default.
• W3084111603 hasConceptScore W3084111603C2778271842 @default.
• W3084111603 hasConceptScore W3084111603C2779134260 @default.
• W3084111603 hasConceptScore W3084111603C2780479503 @default.
• W3084111603 hasConceptScore W3084111603C2781118164 @default.
• W3084111603 hasConceptScore W3084111603C71924100 @default.
• W3084111603 hasFunder F4320323654 @default.
• W3084111603 hasLocation W30841116031 @default.
• W3084111603 hasLocation W30841116032 @default.
• W3084111603 hasLocation W30841116033 @default.

• next