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- W3085208544 abstract "Interleukin (IL)-35 belongs to the IL-12 cytokine family and is a heterodimer of the p35 and Epstein-Barr virus-induced gene 3 (EBI3) subunits. Functionally, IL-35 can promote the proliferation and activation of regulatory T cells (Tregs) and suppress the function of T helper 17 (Th17) cells and other inflammatory cells to inhibit immune responses. In recent years, an abnormal IL-35 expression causing a Th17/Treg imbalance has been associated with the development and progression of several connective tissue diseases (CTDs), such as rheumatoid arthritis (RA), systemic lupus erythematosus (SLE), systemic sclerosis (SSc), dermatomyositis (DM)/polymyositis (PM), and primary Sjogren's syndrome (pSS). Here, we review the role of IL-35 in regulating the balance of Th17/Treg responses in different types of CTDs and provide new insights into the role of IL-35 in these diseases." @default.
- W3085208544 created "2020-09-21" @default.
- W3085208544 creator A5016713360 @default.
- W3085208544 creator A5026262120 @default.
- W3085208544 date "2020-09-11" @default.
- W3085208544 modified "2023-10-17" @default.
- W3085208544 title "Interleukin‐35 regulates the balance of Th17 and Treg responses during the pathogenesis of connective tissue diseases" @default.
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- W3085208544 doi "https://doi.org/10.1111/1756-185x.13962" @default.
- W3085208544 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/32918357" @default.
- W3085208544 hasPublicationYear "2020" @default.
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