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- W3085635562 abstract "Calorie restriction in experimental models, at adolescence, malprograms to metabolic impairments, like that it happens in intrauterine rat models. We aimed to assess the leptin signaling pathway in the hypothalamus in both adolescent and adult rats that were malnourished at puberty. At 30-days-old, Wistar male rats were fed a low-protein diet (4%, LP group) until 60-days-old and then, to dietary rehabilitation, were fed a normal-protein diet (20.5%) from 60- to 120-days-old. Control rats (NP group) were fed a normal-protein diet throughout life. Body weight and food intake were evaluated throughout experimental period. At 60- and 120-days-old, a batch of rats was euthanized to remove visceral fat pad (mesenteric fat), blood and hypothalamus to quantify body fat deposition by biometrical analysis, leptinemia by ELISA and leptin signaling by western blot. Experimental procedures were approved by the research ethical committee for use of animals from the Universidade Federal de Mato Grosso (protocol: 23108.709618/2015-21). All data were analyzed by Student t test, through Graph Pad Prism, for Windows. At 60-days-old, after starvation period, LP-rats showed smaller body weight (–72%, P 0.05). Obesity development in adult rats, malnourished at puberty, is not due to central leptin resistance, but possibly by other neuroendocrine factors that can be affecting energy balance." @default.
- W3085635562 created "2020-09-21" @default.
- W3085635562 creator A5054435557 @default.
- W3085635562 date "2018-10-10" @default.
- W3085635562 modified "2023-09-26" @default.
- W3085635562 title "OBESITY ONSET DUE TO LOW-PROTEIN DIET IN ADOLESCECE IS NOT ASSOCIATED WITH HYPOTHALAMIC LEPTIN RESISTANCE" @default.
- W3085635562 hasPublicationYear "2018" @default.
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