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- W3087762178 endingPage "H1020" @default.
- W3087762178 startingPage "H1008" @default.
- W3087762178 abstract "Chronic inflammatory disorders, including rheumatoid arthritis (RA), are associated with a twofold increase in the incidence of sudden cardiac death (SCD) compared with the healthy population. Although this is partly explained by an increased prevalence of coronary artery disease, growing evidence suggests that ischemia alone cannot completely account for the increased risk. The present review explores the mechanisms of cardiac electrophysiological remodeling in response to chronic inflammation in RA. In particular, it focuses on the roles of nonischemic structural remodeling, altered cardiac ionic currents, and autonomic nervous system dysfunction in ventricular arrhythmogenesis and SCD. It also explores whether common genetic elements predispose to both RA and SCD. Finally, it evaluates the potential dual effects of disease-modifying therapy in both diminishing and promoting the risk of ventricular arrhythmias and SCD." @default.
- W3087762178 created "2020-09-25" @default.
- W3087762178 creator A5011063805 @default.
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- W3087762178 creator A5063900168 @default.
- W3087762178 creator A5071551127 @default.
- W3087762178 creator A5075213080 @default.
- W3087762178 date "2020-11-01" @default.
- W3087762178 modified "2023-10-16" @default.
- W3087762178 title "Proarrhythmic electrophysiological and structural remodeling in rheumatoid arthritis" @default.
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