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- W3091608678 abstract "Depression as illness of civilization is spreading very extensively in last three decades. It is pronounced trend that this disease take up one from top in general structure of diseases. Comparatively mild therapeutic activity in the treatment of depression is explaining by lack of clear apprehensions about pathogenesis of depression. The monoaminergic hypothesis of pathogenesis of depression exhausted overall the possibilities of treatment of depression. The neurotrophic and glutamatergic hypothesis of pathogenesis of depression developed at 90-th year-end — at the first of the 2000-th years expanded pharmacotherapeutic possibilities of desease treatment. However, the mechanisms of pathogenesis of depression mediated by γ-aminobutyric acid were developed poorly. It was viewed participation of receptors of γ-aminobutyric acid types A and B of brain limbic structures neurons in the regulation of emotion and in pathogenesis of anxiety and mood disorders in this review. It was discussed the possibilities of GABA receptor ligands usage as psychopharmacologic drugs for treatment of depression." @default.
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- W3091608678 date "2020-07-19" @default.
- W3091608678 modified "2023-09-26" @default.
- W3091608678 title "ГАМКЕРГИЧЕСКИЕ МЕХАНИЗМЫ ПАТОГЕНЕЗА И ЛЕЧЕНИЯ ДЕПРЕССИВНОГО СИНДРОМА" @default.
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