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- W3091677072 abstract "The relationship between bronchopulmonary Pseudomonas aeruginosa and chronic lung allograft dysfunction (CLAD) has often stimulated considerations of primacy. Which came first, the chicken or the egg? Unlike binary logic, natural circumstances are more complex and demand a pluralistic view of causality, notwithstanding the recognition that in systems of coexistence, mitigation of deleterious outcomes can still be achieved by reducing one of the driving factors in the equation. So it is with airway disease, both large and small, after lung transplantation [1, 2]. Mucosal and structural damage promote an unhealthy local milieu with impaired barrier function, compromised local immunity and reduced efficacy of mucociliary transport. Perturbations of the pulmonary microbiome along with chemical damage from often occult gastric aspiration set the foundation for such a scenario. However, unlike fibrotic luminal occlusion of small airways typical of obliterative bronchiolitis, which is permanent, the damage described above may be amenable to change [3]. Univariable analysis supports attempts at “eradicating” Pseudomonas from bronchopulmonary secretions after lung transplantation <https://bit.ly/2Nw6h5P>" @default.
- W3091677072 created "2020-10-08" @default.
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- W3091677072 date "2020-10-01" @default.
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- W3091677072 title "<i>Pseudomonas</i> and risk factor mitigation for chronic lung allograft dysfunction" @default.
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- W3091677072 doi "https://doi.org/10.1183/13993003.01968-2020" @default.
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