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- W3092433209 abstract "Neutrophil extravasation is a critical event during immune responses to infection or injury that ensures survival. However, given the destructive potential of neutrophil effector molecules such as proteases and reactive oxygen species, strict control of neutrophil recruitment is required to avoid tissue damage. Neutrophil extravasation is a multistep process of adhesive interactions between neutrophils and components of the venular wall, that is, endothelium, basement membrane and pericytes. A plethora of proteins have been identified as critical regulators of each step that provide for compensatory mechanisms thus ensuring that extravasation can happen even if one mechanism is defective. Here, we discuss recent discoveries of how extravasation is regulated with emphasis on neutrophil-specific mechanisms; and discuss potential implications for future research." @default.
- W3092433209 created "2020-10-15" @default.
- W3092433209 creator A5008700363 @default.
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- W3092433209 date "2021-02-01" @default.
- W3092433209 modified "2023-10-16" @default.
- W3092433209 title "The extravasation cascade revisited from a neutrophil perspective" @default.
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- W3092433209 doi "https://doi.org/10.1016/j.cophys.2020.09.014" @default.
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