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• W3107700148 abstract "Abstract COVID-19 has left mankind desperately seeking how to manage dramatically rising infection rates associated with severe disease progressions. COVID-19 courses range from mild symptoms up to multiple organ failure and death, triggered by excessively high serum cytokine levels (IL 1β, IL 6, TNF α, IL 8). The vagally driven cholinergic anti-inflammatory pathway (CAP) stops the action of nuclear factor κB (NF-κB), the transcriptional factor of pro-inflammatory cytokines. Thus, well-balanced cytokine release depends on adequate vagal signaling. Coronaviruses replicate using NF-κB transcriptional factor as well. By degrading the cytoplasmatic inhibitor of NF-κB subunits (IκB), coronaviruses induce unrestricted NF-κB expression accelerating both, virus replication and cytokine transcription. We hypothesize that CAP detriment due to depressed vagal tone critically determines the severity of COVID-19." @default.
• W3107700148 created "2020-12-07" @default.
• W3107700148 creator A5004468488 @default.
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• W3107700148 creator A5089530597 @default.
• W3107700148 date "2020-11-24" @default.
• W3107700148 modified "2023-10-16" @default.
• W3107700148 title "Autonomic balance determines the severity of COVID-19 courses" @default.
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• W3107700148 doi "https://doi.org/10.1186/s42234-020-00058-0" @default.
• W3107700148 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/7683278" @default.
• W3107700148 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/33292846" @default.
• W3107700148 hasPublicationYear "2020" @default.
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