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- W3108592143 abstract "The innate immune system, the first line of defense against pathogens and host tissue damage, initiates pro-inflammatory responses which, when dysregulated, promote inflammation to drive a broad range of autoimmune diseases. Immunomodulatory therapies have been developed to successfully treat several autoimmune diseases, but still many others lack effective treatments. Here, we explore recent advances in how the innate immune system contributes to autoinflammation, from the innate immune sensors that initiate immune responses to how this system regulates the activation of programmed cell death pathways including pyroptosis, apoptosis, necroptosis, and PANoptosis, which involves machinery from the pyroptotic, apoptotic, and necroptotic pathways. Recent advances in our understanding of innate immunity raise important considerations for developing new inflammatory disease treatments that target innate immune signaling and programmed cell death pathways." @default.
- W3108592143 created "2020-12-07" @default.
- W3108592143 creator A5056812960 @default.
- W3108592143 creator A5074251335 @default.
- W3108592143 date "2020-12-01" @default.
- W3108592143 modified "2023-10-14" @default.
- W3108592143 title "The innate immune system and cell death in autoinflammatory and autoimmune disease" @default.
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- W3108592143 doi "https://doi.org/10.1016/j.coi.2020.10.013" @default.
- W3108592143 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/33242752" @default.
- W3108592143 hasPublicationYear "2020" @default.
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