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- W3109330936 endingPage "434" @default.
- W3109330936 startingPage "434" @default.
- W3109330936 abstract "The involvement of inflammation in cancer progression has been the subject of research for many years. Inflammatory milieu and immune response are associated with cancer progression and recurrence. In different types of tumors, growth and metastatic phenotype characterized by the epithelial mesenchymal transition (EMT) process, stemness, and angiogenesis, are increasingly associated with intrinsic or extrinsic inflammation. Among the inflammatory mediators, prostaglandin E2 (PGE2) supports epithelial tumor aggressiveness by several mechanisms, including growth promotion, escape from apoptosis, transactivation of tyrosine kinase growth factor receptors, and induction of angiogenesis. Moreover, PGE2 is an important player in the tumor microenvironment, where it suppresses antitumor immunity and regulates tumor immune evasion, leading to increased tumoral progression. In this review, we describe the current knowledge on the pro-tumoral activity of PGE2 focusing on its role in cancer progression and in the regulation of the tumor microenvironment." @default.
- W3109330936 created "2020-12-07" @default.
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- W3109330936 creator A5048975636 @default.
- W3109330936 creator A5075353845 @default.
- W3109330936 creator A5075916964 @default.
- W3109330936 date "2020-12-01" @default.
- W3109330936 modified "2023-10-16" @default.
- W3109330936 title "Prostaglandin E2 and Cancer: Insight into Tumor Progression and Immunity" @default.
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