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- W3112604045 endingPage "620" @default.
- W3112604045 startingPage "606" @default.
- W3112604045 abstract "Successful treatment of tuberculosis (TB) depends on the eradication of its causative agent Mycobacterium tuberculosis (Mtb) in the host. However, the emergence of phenotypically drug-resistant Mtb in the host environment tempers the ability of antibiotics to cure disease. Host immunity produces diverse microenvironmental niches that are exploited by Mtb to mobilize adaptation programs. Such differential interactions amplify pre-existing heterogeneity in the host-pathogen milieu to influence disease pathology and therapy outcome. Therefore, comprehending the intricacies of phenotypic heterogeneity can be an empirical step forward in potentiating drug action. With this goal, we review the interconnectedness of the lesional, cellular, and bacterial heterogeneity underlying phenotypic drug resistance. Based on this information, we anticipate the development of new therapeutic strategies targeting host-pathogen heterogeneity to cure TB." @default.
- W3112604045 created "2020-12-21" @default.
- W3112604045 creator A5037644194 @default.
- W3112604045 creator A5044886937 @default.
- W3112604045 creator A5058504876 @default.
- W3112604045 creator A5067909461 @default.
- W3112604045 date "2021-07-01" @default.
- W3112604045 modified "2023-10-14" @default.
- W3112604045 title "Heterogeneous Host–Pathogen Encounters Coordinate Antibiotic Resilience in Mycobacterium tuberculosis" @default.
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