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• W3120481800 abstract "The syndrome of juxtaglomerular hyperplasia with hypokalemic, hypochloremic alkalosis, aldosteronism, hyperreninemia, and normal blood pressure may be familial, and is probably inherited as a recessive trait. It can usually be distinguished from salt-losing chronic glomerulo-nephritis by the histologic appearance of the kidneys, and by the absence of sodium “leak” with a low-sodium intake. Urinary, and thus renal, prostaglandin E is increased in the untreated patient: when this is lowered with prostaglandin synthetase inhibitors, the plasma renin and aldosterone decrease, and the plasma potassium concentration rises. These and other results suggest a partial control of renin secretion by prostaglandin E, and also suggest that prostaglandin E is an essential feature of the syndrome; it may, indeed, be a “proximal” cause of all the essential features." @default.
• W3120481800 created "2021-01-18" @default.
• W3120481800 creator A5028639967 @default.
• W3120481800 date "1977-06-01" @default.
• W3120481800 modified "2023-09-27" @default.
• W3120481800 title "Bartter's Syndrome" @default.
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• W3120481800 doi "https://doi.org/10.1016/s0094-0143(21)01042-9" @default.
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