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- W3121562625 endingPage "651" @default.
- W3121562625 startingPage "643" @default.
- W3121562625 abstract "The pathophysiological mechanisms of neuroinflammation, angiogenesis, and neuroplasticity are currently the hotspots of researches in ischemic stroke. Regulatory T cells (Tregs), a subset of T cells that control inflammatory and immune responses in the body, are closely related to the pathogenesis of ischemic stroke. They participate in the inflammatory response and neuroplasticity process of ischemic stroke by various mechanisms, such as secretion of anti-inflammatory factors, inhibition of pro-inflammatory factors, induction of cell lysis, production of the factors that promote neural regeneration, and modulation of microglial and macrophage polarization. However, it remains unclear whether Tregs play a beneficial or deleterious role in ischemic stroke and the effect of Tregs in different stages of ischemic stroke. Here, we discuss the dynamic changes of Tregs at various stages of experimental and clinical stroke, the potential mechanisms under Tregs in regulating stroke and the preclinical studies of Tregs-related treatments, in order to provide a reference for clinical treatment." @default.
- W3121562625 created "2021-02-01" @default.
- W3121562625 creator A5009988205 @default.
- W3121562625 creator A5021522886 @default.
- W3121562625 creator A5048445024 @default.
- W3121562625 creator A5072097629 @default.
- W3121562625 creator A5077128913 @default.
- W3121562625 creator A5085642634 @default.
- W3121562625 date "2021-01-20" @default.
- W3121562625 modified "2023-10-10" @default.
- W3121562625 title "Regulatory T cells in ischemic stroke" @default.
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