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- W3121797937 endingPage "161" @default.
- W3121797937 startingPage "161" @default.
- W3121797937 abstract "Respiratory viruses such as influenza and severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) are a constant threat to public health given their ability to cause global pandemics. Infection with either virus may lead to aberrant host responses, such as excessive immune cell recruitment and activation, dysregulated inflammation, and coagulopathy. These may contribute to the development of lung edema and respiratory failure. An increasing amount of evidence suggests that lung endothelial cells play a critical role in the pathogenesis of both viruses. In this review, we discuss how infection with influenza or SARS-CoV-2 may induce endothelial dysfunction. We compare the effects of infection of these two viruses, how they may contribute to pathogenesis, and discuss the implications for potential treatment. Understanding the differences between the effects of these two viruses on lung endothelial cells will provide important insight to guide the development of therapeutics." @default.
- W3121797937 created "2021-02-01" @default.
- W3121797937 creator A5009231300 @default.
- W3121797937 creator A5035664128 @default.
- W3121797937 date "2021-01-22" @default.
- W3121797937 modified "2023-09-23" @default.
- W3121797937 title "Interactions of Influenza and SARS-CoV-2 with the Lung Endothelium: Similarities, Differences, and Implications for Therapy" @default.
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