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- W3127016763 endingPage "45" @default.
- W3127016763 startingPage "37" @default.
- W3127016763 abstract "The vascular endothelium functions as a selective barrier between the circulation and body tissues, mediating multiple signal transduction events involved in the acute inflammatory response, angiogenesis, and the regulation of vasomotor tone. The endothelium generates and responds to a variety of signaling molecules; prominent among these are reactive oxygen species (ROS). ROS are oxygen-containing compounds such as hydrogen peroxide (H2O2), superoxide (O2•−), hydroxyl radical (•OH), and peroxynitrite (ONOO−) generated by the partial or complete reduction of molecular oxygen (O2). Historically, ROS have been described as injurious byproducts of cellular metabolism that are produced in excess in the vascular endothelium during myocardial infarction, cardiovascular and metabolic disease, ischemic stroke, and organ transplantation, contributing to cell death and tissue injury. Work in recent years, however, has begun to identify critical signaling and regulatory roles of certain ROS in the healthy endothelium. Generally, the species generated, the site (intracellular compartment, cell type, and tissue), and the scale of generation likely dictate whether ROS participate in cellular signaling or contribute to oxidative damage." @default.
- W3127016763 created "2021-02-15" @default.
- W3127016763 creator A5027325862 @default.
- W3127016763 creator A5059302902 @default.
- W3127016763 date "2021-01-01" @default.
- W3127016763 modified "2023-10-16" @default.
- W3127016763 title "Reactive oxygen species, redox signaling, and regulation of vascular endothelial signaling" @default.
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