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- W3127506782 endingPage "1539" @default.
- W3127506782 startingPage "1519" @default.
- W3127506782 abstract "Nonalcoholic fatty liver disease comprises a wide spectrum of liver injuries from simple steatosis to steatohepatitis and cirrhosis. Nonalcoholic steatohepatitis (NASH) is defined when liver steatosis is associated with inflammation, hepatocyte damage, and fibrosis. A genetic predisposition and environmental insults (ie, dietary habits, obesity) are putatively responsible for NASH progression. Here, we present the impact of the lipid-sensing nuclear receptors in the pathogenesis and treatment of NASH. In detail, we discuss the pros and cons of the putative transcriptional action of the fatty acid sensors (peroxisome proliferator-activated receptors), the bile acid sensor (farnesoid X receptor), and the oxysterol sensor (liver X receptors) in the pathogenesis and bona fide treatment of NASH." @default.
- W3127506782 created "2021-02-15" @default.
- W3127506782 creator A5009453928 @default.
- W3127506782 creator A5041761706 @default.
- W3127506782 creator A5089277303 @default.
- W3127506782 date "2021-01-01" @default.
- W3127506782 modified "2023-10-15" @default.
- W3127506782 title "Transcriptional Regulation of Metabolic Pathways via Lipid-Sensing Nuclear Receptors PPARs, FXR, and LXR in NASH" @default.
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