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- W3129808436 endingPage "1956" @default.
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- W3129808436 abstract "Bcl-xL represents a family of proteins responsible for the regulation of the intrinsic apoptosis pathway. Due to its anti-apoptotic activity, Bcl-xL co-determines the viability of various virally infected cells. Their survival may determine the effectiveness of viral replication and spread, dynamics of systemic infection, and viral pathogenesis. In this paper, we have reviewed the role of Bcl-xL in the context of host infection by eight different RNA and DNA viruses: hepatitis B virus (HBV), hepatitis C virus (HCV), human immunodeficiency virus (HIV), influenza A virus (IAV), Epstein-Barr virus (EBV), human T-lymphotropic virus type-1 (HTLV-1), Maraba virus (MRBV), Schmallenberg virus (SBV) and coronavirus (CoV). We have described an influence of viral infection on the intracellular level of Bcl-xL and discussed the impact of Bcl-xL-dependent cell survival control on infection-accompanying pathogenic events such as tissue damage or oncogenesis. We have also presented anti-viral treatment strategies based on the pharmacological regulation of Bcl-xL expression or activity." @default.
- W3129808436 created "2021-03-01" @default.
- W3129808436 creator A5049118687 @default.
- W3129808436 creator A5074950573 @default.
- W3129808436 creator A5083154102 @default.
- W3129808436 creator A5084515790 @default.
- W3129808436 date "2021-02-16" @default.
- W3129808436 modified "2023-10-03" @default.
- W3129808436 title "The Role of Bcl-xL Protein in Viral Infections" @default.
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