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- W3133674390 endingPage "480" @default.
- W3133674390 startingPage "465" @default.
- W3133674390 abstract "Severe acute respiratory syndrome coronavirus-2 is a positive-sense RNA virus, a causal agent of ongoing COVID-19 pandemic. ACE2R methylation across three CpG sites (cg04013915, cg08559914, cg03536816) determines the host cell’s entry. It regulates ACE2 expression by controlling the SIRT1 and KDM5B activity. Further, it regulates Type I and III IFN response by modulating H3K27me3 and H3K4me3 histone mark. SARS-CoV-2 protein with bromodomain and protein E mimics bromodomain histones and evades from host immune response. The 2′-O MTases mimics the host’s cap1 structure and plays a vital role in immune evasion through Hsp90-mediated epigenetic process to hijack the infected cells. Although the current review highlighted the critical epigenetic events associated with SARS-CoV-2 immune evasion, the detailed mechanism is yet to be elucidated." @default.
- W3133674390 created "2021-03-15" @default.
- W3133674390 creator A5009928146 @default.
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- W3133674390 creator A5054422735 @default.
- W3133674390 creator A5061845672 @default.
- W3133674390 creator A5062968938 @default.
- W3133674390 creator A5088271731 @default.
- W3133674390 date "2021-03-01" @default.
- W3133674390 modified "2023-10-14" @default.
- W3133674390 title "An immune epigenetic insight to COVID-19 infection" @default.
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