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- W3133852020 abstract "ATM is often dubbed the master regulator of the DNA double stranded break (DSB) response. Since proper induction and repair of DNA DSBs forms the core of immunological diversity, it is surprising that patients with ataxia telangiectasia generally have a mild immunodeficiency in contrast to other DSB repair syndromes. In this review, we address this discrepancy by delving into the functions of ATM in DSB repair and cell cycle control and translate these to adaptive immunity. We conclude that ATM, despite its myriad functions, is not an absolute requirement for acquiring sufficient levels of immunological diversity to prevent severe viral and opportunistic infections. There is, however, a more clinically pronounced antibody deficiency in ataxia telangiectasia due to disturbed class switch recombination." @default.
- W3133852020 created "2021-03-15" @default.
- W3133852020 creator A5043968197 @default.
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- W3133852020 creator A5060357125 @default.
- W3133852020 creator A5084443015 @default.
- W3133852020 date "2021-04-01" @default.
- W3133852020 modified "2023-10-16" @default.
- W3133852020 title "ATM: Translating the DNA Damage Response to Adaptive Immunity" @default.
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