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• W3134901541 endingPage "577542" @default.
• W3134901541 startingPage "577542" @default.
• W3134901541 abstract "We hypothesized that humoral immunity stimulation in the CNS in HIV-1C patients would be lower than that in HIV-1B due to a defective Tat chemokine dimotif (C30C31) that might influence cellular trafficking and CNS inflammation. Sixty-eight paired CSF and blood samples from people with HIV (PWH), free of CNS opportunistic infections, were included, HIV-1B (n = 27), HIV-1C (n = 26), and HIV negative (n = 25). IgG intrathecal synthesis was assayed using quantitative and qualitative methods. IgG oligoclonal bands (OCB) in CSF were observed in 51% of PWH, comparable between HIV-1B and HIV-1C, as well as the medians of IgG intrathecal synthesis formulas. The group with HIV infection aviremic in CSF and blood showed 75% of OCB. There was a poor positive correlation between the IgG quotient and GDS. The impact of HIV-1 on IgG intrathecal production was not subtype dependent. Low-grade CNS intrathecal IgG production persists in HIV CNS infection even in PWH with CSF and blood HIV RNA controlled." @default.
• W3134901541 created "2021-03-15" @default.
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• W3134901541 date "2021-06-01" @default.
• W3134901541 modified "2023-10-18" @default.
• W3134901541 title "IgG intrathecal synthesis in HIV-associated neurocognitive disorder (HAND) according to the HIV-1 subtypes and pattern of HIV RNA in CNS and plasma compartments" @default.
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• W3134901541 doi "https://doi.org/10.1016/j.jneuroim.2021.577542" @default.
• W3134901541 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/8102367" @default.
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