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- W313660276 abstract "Publisher Summary This chapter discusses the possible role of slow sodium channel inactivation in regulating membrane excitability in mammalian skeletal muscle. Three different voltage clamp methods have been used to study sodium currents in adult skeletal muscle fibers, which include three-microelectrode, Vaseline-gap or sucrose-gap, and loose-patch. Slow inactivation (SI) is present at the resting potential of mammalian muscle fibers, and SI acts at more negative membrane potentials in fast than in slow twitch fibers. SI probably results from a membrane potential dependent reversible conformation change of the sodium channel molecule to a nonconducting state in a variety of excitable membranes. There is no evidence that the conductance of open sodium channels varies in a continuous fashion with membrane potential. SI probably represents a gradual change in the number of excitable sodium channels rather than a change in the conductance of individual channels. A steady potential applied to the pipette tip not only changes the membrane potential under the patch but also creates a lateral electric field under the pipette rim." @default.
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- W313660276 date "1988-01-01" @default.
- W313660276 modified "2023-09-27" @default.
- W313660276 title "The Possible Role of Slow Sodium Channel Inactivation in Regulating Membrane Excitability in Mammalian Skeletal Muscle" @default.
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- W313660276 doi "https://doi.org/10.1016/b978-0-409-90093-4.50019-5" @default.
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