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- W3138133674 abstract "Epithelial-mesenchymal transition (EMT) is a dynamic process by which epithelial cells loss their phenotype and acquire mesenchymal traits, including increased migratory and invasive capacities. EMT is involved in physiological processes, such as embryogenesis and wound healing, and in pathological processes such as cancer, playing a pivotal role in tumor progression and metastasis. Pituitary tumors, although typically benign, can be locally invasive. Different studies have shown the association of EMT with increased tumor size and invasion in pituitary tumors, and in particular with a poor response to Somatostatin Receptor Ligands (SRLs) treatment in GH-producing pituitary tumors, the main cause of acromegaly. This review will summarize the current knowledge regarding EMT and SRLs resistance in acromegaly and, based on this relation, will suggest new biomarkers and possible therapies to SRLs resistant tumors." @default.
- W3138133674 created "2021-03-29" @default.
- W3138133674 creator A5000731211 @default.
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- W3138133674 creator A5074675449 @default.
- W3138133674 creator A5089216576 @default.
- W3138133674 date "2021-03-15" @default.
- W3138133674 modified "2023-10-16" @default.
- W3138133674 title "Epithelial–Mesenchymal Transition in the Resistance to Somatostatin Receptor Ligands in Acromegaly" @default.
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- W3138133674 doi "https://doi.org/10.3389/fendo.2021.646210" @default.
- W3138133674 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/8006574" @default.
- W3138133674 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/33790868" @default.