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- W3139197788 startingPage "103277" @default.
- W3139197788 abstract "Acute myeloid leukemia (AML) remains the most lethal of leukemias and a small population of cells called leukemic stem cells (LSCs) has been associated with disease relapses. Some cell signaling pathways play an important role in AML survival, proliferation and self-renewal properties and are abnormally activated or suppressed in LSCs. This includes the NF-κB, Wnt/β-catenin, Hedgehog, Notch, EGFR, JAK/STAT, PI3K/AKT/mTOR, TGF/SMAD and PPAR pathways. This review aimed to discuss these pathways as molecular targets for eliminating AML LSCs. Herein, inhibitors/activators of these pathways were summarized as a potential new anti-AML therapy capable of eliminating LSCs to guide future researches. The clinical use of cell signaling pathways data can be useful to enhance the anti-AML therapy." @default.
- W3139197788 created "2021-03-29" @default.
- W3139197788 creator A5022743464 @default.
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- W3139197788 creator A5063241194 @default.
- W3139197788 creator A5073986792 @default.
- W3139197788 creator A5076970280 @default.
- W3139197788 date "2021-04-01" @default.
- W3139197788 modified "2023-10-01" @default.
- W3139197788 title "Cell signaling pathways as molecular targets to eliminate AML stem cells" @default.
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