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• W3140629615 abstract "Cullin 戒指 E3 ligases (CRL ) 调整植物 developmentand 的不同方面被他们的 cullin 子单元的修正与象 ubiquitin 一样蛋白质 NEDD8 激活(表示的神经先锋房间发展地下面调整 8 )(neddylation ) 并且由 NEDD8 移动(deneddylation ) 撤销了。CONSTITUTIVELY PHOTOMORPHOGENIC9 (COP9 ) signalosome (CSN ) 行为作为由回复的 CRLsactivity 的一个分子的开关，他们的 neddylation 地位，而是它对胚胎、早的幼苗开发的贡献仍然保持糟糕描绘了。这里，我们分析了 csn 异种的 phenotypic 缺点并且在胚胎、早的幼苗开发期间监视了 cullin deneddylation/neddylation 比率。Weshow 当 csn 异种能完成 embryogenesis 时(以更慢的步的虽然比野类型) 并且能发芽(以减少的率的虽然) ，他们日益增多地在 germinationuntil 之上失去分裂组织活动他们变得不能支撑生长。我们也证明 cullin 蛋白质的多数是日益增多地 neddylated 在在种子萌芽之上的种子成熟和 becomedeneddylated 的迟了的阶段期间。在 cullin neddylation 地位的这发展地调整的移动在 csn 异种是不在的。我们断定 CSN 和它的 cullin deneddylation 活动被要求在 Arabidopsis 支撑 postembryonic 分裂组织功能。" @default.
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• W3140629615 date "2015-01-01" @default.
• W3140629615 modified "2023-10-12" @default.
• W3140629615 title "The COP9 SIGNALOSOME Is Required for Postembryonic Meristem Maintenance in Arabidopsis thaliana" @default.
• W3140629615 hasPublicationYear "2015" @default.
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