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• W3140734933 abstract "为了调查(FGF-2 ) 成纤维细胞生长因素 2 在人的小房间肺癌症房间线 H446 由禁止 apoptosis 的机制，使遭到了到浆液饥饿， apoptosis 被流动 cytometry，染色的 Hoechst 33258， caspase-3 活动，和 DNA 破碎评估。FGF-2 和蛋白质 kinase C (PKC ) translocation 导致的 Survivin 表示被 subcellular 分别和西方的污点分析检测。另外，到细胞质的从线粒体的 Smac 的 FGF-2-in-duced 版本被西方的弄污和 immunofluorescence 分析。FGF-2 减少了浆液饥饿和起来调整的 survivin 表示在剂量依赖者 andtime-dependentmanner 在 H446 房间导致的 apoptosis， andinhibitedcaspase-3 活动。FGF-2 也从线粒体禁止了 Smac 的版本到浆液饥饿导致并且从细胞质增加了 PKC translocation 到房间膜的细胞质。另外， PKC 禁止者禁止了 survivin 的表示。FGF-2 起来调整在 H446 房间和块的 survivin 蛋白质的表示到细胞质的从线粒体的 Smac 的版本。PKC 调整了 FGF-2-induced survivin 表示。因此， survivin， Smac，和 PKC 可能在人的小房间肺癌症房间由 FGF-2 在 apoptosis 的抑制起重要作用。" @default.
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• W3140734933 date "2008-01-01" @default.
• W3140734933 modified "2023-09-26" @default.
• W3140734933 title "Inhibition of fibroblast growth factor 2-induced apoptosis involves survivin expression, protein kinase Cα activation and subcellular translocation of Smac in human small cell lung cancer cells" @default.
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