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- W3140989704 abstract "目的将在 vivo 在老鼠在长期的 potentiation (LTP ) 和 AMPA 受体子单元上探索暴露的效果到铝(艾尔) 。aluminum-maltolate 建筑群的方法不同剂量[艾尔(mal ) 3 ] 经由尖锐 intracerebroventricular (i.c.v ) 被给老鼠注射和 subchronic intraperitoneal (i.p ) 注射。后面的艾尔暴露,海马趾的 LTP 被地 potentiation 技术从老鼠马头鱼尾的怪兽的 CA1 区域在全部、充实膜的摘录在 vivo 和 AMPAR 子单元蛋白质(GluR1 和 GluR2 ) 的表示记录被西方的污点试金检测。敏锐的艾尔处理生产了的结果在老鼠马头鱼尾的怪兽和在膜的 GluR1 和 GluR2 的剂量依赖者减少的 LTP 的剂量依赖者抑制提取;然而,没有类似的变化在全部的房间摘录被发现,它在马头鱼尾的怪兽从细胞内部的水池建议 AMPA 受体子单元的减少的 trafficking 到 synaptic 地点。LTP 和在膜并且在全部的摘录的 AMPA 受体子单元的表示上的镇压效果在 subchronic 艾尔处理以后被发现的剂量依赖者,在子单元的表示从细胞内部的水池在 AMPA 受体子单元 trafficking 显示减少到 synaptic 地点和另外的减小。结论艾尔(mal ) 在老鼠的 3 显然并且 dose-dependently 压制的 LTP 在 vivo 的海马趾的 CA1 区域,和这抑制可能在 AMPA 受体子单元蛋白质的表示与 trafficking 和减少有关。然而,位于这些观察下面的机制需要推进调查。" @default.
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- W3140989704 date "2014-01-01" @default.
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- W3140989704 title "Effects of Exposure to Aluminum on Long-term Potentiation and AMPA Receptor Subunits in Rats in vivo" @default.
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