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- W3141400888 abstract "BACKGROUNDRecent studies examining mechanisms of defibrillation have focused on prolongation of graded cellular response duration during refractory period stimulation. This mechanism assumes that defibrillation shocks interact with ventricular cells during the process of repolarization.METHODS AND RESULTSTo test this assumption, we examined monophasic action potentials (MAPs) from 171 episodes of induced ventricular dysrhythmia associated with loss of systemic perfusion pressure in 22 patients undergoing nonthoracotomy defibrillator implantation. Ventricular fibrillation (VF)/polymorphic ventricular tachycardia (PVT), defined by an irregular limb lead I morphology, was present in 156 dysrhythmia episodes. Monomorphic ventricular tachycardia (VT), present in the remaining 15 episodes, was associated with regular limb lead morphology. All episodes were examined for MAP cycle length, variation, fractionation, and repolarization. VF/PVT cycle length was 215 +/- 28 msec, with a 14 +/- 7% (33 +/- 20-msec) cycle..." @default.
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- W3141400888 date "1993-01-01" @default.
- W3141400888 modified "2023-09-24" @default.
- W3141400888 title "Characterization of ventricular fibrillation based on monophasic action potential morphology in the human heart." @default.
- W3141400888 hasPublicationYear "1993" @default.
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