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- W3141411322 abstract "Microglial 激活在许多神经病学的疾病被含有。在这研究，我们检验了粉防已硷( TET )的效果， microglial activation.Methods 上的中国植物 Stephania tetrandra S 穆尔的主要pharmacologically活跃的混合物:微神经胶质 pretreated 与或没有 TET 被脂肪的多糖( LPS )在 vitro.Nitric 氧化物激活(没有)版本，超级氧化物阴离子(O2~-)产生，象 TNF 一样--由微神经胶质的and interleukin-6 ( IL-6 )生产被测量 afterwards.Electrophoretic 活动性移动试金被执行决定在微神经胶质的 NF-KB 活动是否被 TET treatment.Results 影响:我们发现 TET 由减少禁止了微神经胶质的 导致LPS 的激活生产没有并且O2~，因而在 导致LPS 的 microglial 激活影响 TNF-alpha 和 IL-6 的版本。如此的镇压效果被禁止抄写因素 NF-KAPPAB activation.Conclusion 伴随: 我们的结果建议 TET 可能由禁止 NF-KAPPA 调制导致 LPS 的 microglial 激活煽动性的因素的调停 B 的版本。" @default.
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- W3141411322 date "2008-01-01" @default.
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- W3141411322 title "Tetrandrine suppresses lipopolysaccharide-induced microglial activation by inhibiting NF-κB pathway" @default.
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