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• W3141446706 abstract "Apolipoprotein A-I (ApoA-I) mimetic peptide inhibits the development of atherosclerosis (AS) in apolipo- protein E-deficient mice; however, the underlying mechanism remains unclear. Endothelial progenitor cells (EPCs) can pre- vent AS progression through repairing proatherogenic factors impaired endothelium. In the present study, we examined the effect of reverse D-4F, one of apoA-I mimetic peptide on the proliferation, migration, and tube formation of mouse bone marrow-derived late EPCs. The present study showed that reverse D-4F (10-100 lg/ml) significantly improved the proliferation, migration, and tube formation of EPCs in a dose- dependent manner, and activated phospho-AKT at serine res- idue 473 and phospho-eNOS at serine residue 1177. LY294002 (PI3-kinase inhibitor) and L-NAME (NOS inhibitor) signifi- cantly inhibited reverse D-4F mediated improvement of EPCs biological functions, and LY294002 significantly decreased reverse D-4F stimulated activation of phospho-AKT (473) and phospho-eNOS (1177). The results indicate that reverse D-4F mediated improvement of EPCs functions is dependent on the PI3K/AKT/eNOS pathway." @default.
• W3141446706 created "2021-04-13" @default.
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• W3141446706 date "2013-01-01" @default.
• W3141446706 modified "2023-09-23" @default.
• W3141446706 title "Apolipoprotein A-I mimetic peptide reverse D-4F improves the biological functions of mouse bone marrow-derived late EPCs via PI3K/AKT/eNOS pathway" @default.
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• W3141446706 hasPublicationYear "2013" @default.
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