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- W3142598609 abstract "瞄准：现在的学习试图阐明在在针的 level.METHODS 的神经病的疼痛的致病的 T 子类型钙隧道(Cav3.1， Cav3.2，和 Cav3.3 ) 的角色：背面的根中心(电荷耦合器件) 的长期的压缩老鼠模型被收养。Cav3.1， Cav3.2，和 Cav3.3 或正常的 Theantisense oligonucleotide 盐(NS ) 每从第一天的天是 intrathecallyadministered 两次到在操作以后的第四天。爪机械退却阀值和爪热退却潜伏被测量分别地评估 tactileallodynia 和热痛觉过敏。结果：电荷耦合器件老鼠在操作以后开发了可靠 tactileallodynia 和热痛觉过敏。Cav3.2 和 Cav3.3 的 antisenseoligonucleotide 的 Intrathecal 管理显著地在电荷耦合器件老鼠，然而并非 Cav3.1 减轻了触觉的 allodynia 和热痛觉过敏。结论：在针的绳索的 Cav3.2 和 Cav3.3 子类型钙隧道可以在神经病的疼痛的致病起一个重要作用，它可以贡献神经病的疼痛的管理。" @default.
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- W3142598609 date "2006-01-01" @default.
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- W3142598609 title "Intrathecal administration of Cav3.2 and Cav3.3 antisense oligonucleotide reverses tactile allodynia and thermal hyperalgesia in rats following chronic compression of dorsal root of ganglion" @default.
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