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- W3144501686 abstract "瞄准：调查长期的 H pylori 感染是否有潜力在蒙古的沙鼠模型导致胰腺炎，并且它是否依赖于一个未经触动的类型 IV 分泌物系统。方法：蒙古的沙鼠感染 H pylori 打的野类型(WT ) 我拉紧 B128 或它的 isogenic 异种 B128 三角洲谨慎(有缺点的类型 IV 分泌物) 。在七月感染以后， H pylori 从窦和语料库被重新孤立， H pylori DNA 被嵌套半的聚合酶链反应(PCR ) 分析。发炎和组织学的变化被免疫组织化学在胃的窦，语料库，和胰记录。Cytokine mRNA，胃的 pH，血浆促胃液素，淀粉酶，脂肪分解酵素，和葡萄糖层次是坚定的。结果：H pylori 感染率是 95% 。uninfected 组，发达透壁发炎和慢性胰炎的八个感染的动物，而是没有。邻近窦的胰腺的脑叶的广泛的空隙的纤维变性和发炎被三色的污点证实，并且免疫组织化学地。支持 inflammatory cytokine mRNA 显著地在所有感染的沙鼠的窦粘膜被增加。在语料库，感染 WT 的动物和那些发展中的透壁发炎和胰腺炎的仅仅 cytokine 层次显著地被增加。脂肪分解酵素的层次，然而并非葡萄糖或淀粉酶层次，显著地在胰腺炎组被减少。H pylori DNA 在感染的窦和语料库织物，然而并非在胰被检测。结论：H pylori 感染能独立于类型 IV 分泌物系统在蒙古的沙鼠导致慢性胰炎，可能由与穿透性溃疡联系的间接机制。" @default.
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- W3144501686 date "2007-01-01" @default.
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- W3144501686 title "H pylori infection causes chronic pancreatitis in Mongolian gerbils" @default.
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