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- W3146887489 abstract "目的:到学习，接合的机制在 5-hydroxytryptaminel A/7 (5-HT_( 1A/7 ))包含了心脏的迷走神经 preganglionic ( CVPN )的调停受体的反射控制 .Methods : CVPN retrogradely 在新生的老鼠的脑干片被标记并且识别，并且他们的接合的活动用整个房间的patch-clamp.Results:8-Hydroxy-2-( di-N-propylamino )被检验四氢萘( 8-OH-DPAT )，5-HT_( 1A/7 )的收缩筋受体，没在 CVPN.In 的输入对比的 glutamatergic 上有效果，它显著地减少了频率和自发的禁止的 postsynaptic 水流( sIPSC ) .8-OH-DPAT 也引起了 GABAergic 水流的重要振幅减少的 GABAergic 和 glycinergic 的振幅由原子核道的刺激唤起了我们 solitarius.Both 频率抑制和 GABAergic 和 glycinergic sIPSC 的振幅抑制由 8-OH-DPAT 有剂量依赖者趋势并且能被 WAY-100635 颠倒，5-HT_( 1A/7 )的一个对手 receptors.In 河豚毒素的 pre-exist-ence ， 8-OH-DPAT 没在 GABAergic 或 glycinergic 上有效果缩小的禁止的 postsynaptic 水流，并且 GABAergic 或 glycinergic 水流上的效果都没由外长的伽马氨基丁酸或 glycine.Conclusion 唤起:5-HT_( 1A/7 )受体收缩筋经由 GABAergic 和 glycinergic 输入的抑制间接地使 CVPN 激动。这些调查结果部分地至少表明了接合的机制在未经触动的动物在 5-HT_(1A/7 ) 包含了心脏的迷走神经神经的调停受体的反射控制。" @default.
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- W3146887489 date "2008-01-01" @default.
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- W3146887489 title "5-HT1A/7 receptor agonist excites cardiac vagal neurons via inhibition of both GABAergic and glycinergic inputs" @default.
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