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- W3146894530 abstract "瞄准：在尖锐胰腺炎(AP ) 调查细胞间的粘附 molecule-1 (ICAM-1 ) 和它的相反的受体 LFA-1 和 Mac-1 的角色。方法：SD 老鼠随机被分配到 AP 组和控制组(25 只老鼠每) 。AP 被 5% chenodeoxycholic 酸的注入导致进胰腺的管，由胰腺的管的结扎列在后面。老鼠在胰腺炎的正式就职以后在 1， 3， 6， 12 和 24 h 被牺牲。五只老鼠同时被牺牲在在从胰和肺的血和标本前的二个组的点被获得。浆液淀粉酶和腹水被测量在每次指。在点被免疫组织化学在胰和肺估计的不同时间的 ICAM-1 的表示，和在不同时间点的 neutrophils 上的 LAF-1 和 Mac-1 的表示被流动血细胞计数器检测。结果：AP 的正式就职被淀粉酶和组织学的研究的浆液层次证实。在在根本，时间削尖的控制组(P<0.05 或 P<0.01 ) 比那显著地增加的胰的 ICAM-1 的表示，以及在除了在 1 h 的肺的表示。在血的 neutrophil 上的 LFA-1 和 Mac-1 的表达式在几个次点(P<0.05 或 P<0.01 ) 在控制组比那在 AP 组显著地增加了。腹水的数量和 AP 组的浆液淀粉酶水平比根本，时间削尖的控制组(P<0.05 或 P<0.01 ) 的显著地增加了。到这些的平行结果，重要嗜中性的渗入在胰被发现， AP 的肺纸巾组织老鼠。结论：我们的调查结果在调停为 ICAM-1， LFA-1 和 Mac-1 建议重要角色到全身的病的从局部病的 AP 的发展。ICAM-1， LFA-1， Mac-1 和随后的白血球渗入的 Upregulation 看起来是在 AP 的胰腺、肺的损害的重要事件。" @default.
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- W3146894530 date "2006-01-01" @default.
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- W3146894530 title "Expression and significance of ICAM-1 and its counter receptors LFA-1 and Mac-1 in experimental acute pancreatitis of rats" @default.
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