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- W3146967044 abstract "皮肤接枝拒绝是典型细胞的有免疫力的回答，主要由 T 房间调停了。联系抗原 4-immunoglobin (CTLA4-Ig ) 扩大的细胞毒素的 T 淋巴细胞由堵住 T 房间合作刺激小径并且禁止 T 房间激活的接枝幸存。在延长皮肤接枝幸存调查 CTLA4-Ig 的功效，人的 CTLA4-Ig (hCTLA4-Ig ) 被设计到在在由用 K14 倡导者的 transgenesis 的老鼠皮肤的快车上。反向的抄写聚合酶链反应(RT-PCR ) 和西方的污点试金显示 CTLA4-Ig 的表达式仍然保持与内部控制蛋白质相比皮特定、相对不变， AKT，通过七代。在转基因的老鼠重量的单位的 hCTLA4-Ig 蛋白质的存在和集中一由连接酶的免疫吸着剂试金(ELISA ) 是坚定的，并且结果显示浆液 CTLA4-Ig 集中也通过代仍然保持不变。转基因的老鼠皮肤 grafted 的幸存显著地从一样的老鼠紧张与野类型的皮肤的相比被延长到老鼠创伤上，并且在所有七代之中仍然保持可比较。这建议简历活跃 hCTLA4-Ig 蛋白质稳定地通过至少七代在 transgenical 鼠标被表示，它与稳定的皮特定的 CTLA4-Ig 表达式一致。结果证明在 K14 倡导者驾驶的皮肤的 hCTLA4-Ig 的转基因的表示通过代仍然保持不变，并且一根转基因的线能被建立 reproducibly 向转基因的皮肤提供扩大幸存。" @default.
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- W3146967044 date "2006-01-01" @default.
- W3146967044 modified "2023-09-23" @default.
- W3146967044 title "Stable Skin-specific Overexpression of Human CTLA4-Ig in Transgenic Mice through Seven Generations" @default.
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