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- W3147591489 abstract "瞄准:调查朝鲜槲寄生植物凝血素的反癌症机制(Viscum 相册 coloratum 凝集素, VCA ) 用一个人,结肠癌房间排队(科罗拉多) 。方法:科罗拉多房间上的 VCA 的细胞毒素的效果被 3- 决定(4, 5-dimethylthiazol-2-yl )-2, 5-diphenyltetrazolium 溴化物(MTT ) 试金在试管内和杀死肿瘤的效果在活体内。为了学习,包含的机制,各种各样的 pro-caspases 的表示, anti-apoptotic 蛋白质,和死亡受体被西方的污点决定。决定受体哪个死亡涉及科罗拉多房间的导致 VCA 的 apoptosis,细胞毒性被 MTT 试金术后疗法与死亡受体的收缩筋或对手检验。结果:VCA 在举止和在裸体老鼠的肿瘤的导致的完全的回归与科罗拉多房间移植了的一个时间依赖者和剂量依赖者杀死了科罗拉多房间。有 VCA 的科罗拉多房间的处理包括受体交往蛋白质,原子 factor-kappaB, apoptosis 蛋白质的 X 连接禁止者,和 Akt/protein 激酶 B 激活 caspase-2, -3,-8, 和 anti-apoptotic 分子的减少的表示。我们然后在导致 VCA 的 apoptosis 检验了死亡受体的参与。在检验的死亡受体之中,仅仅肿瘤坏死因素受体 1 涉及科罗拉多房间的 apoptosis,由导致 VCA 的 apoptosis 的抑制证实了并且减少 caspases 的激活,特别地 caspase-8,由肿瘤坏死因素受体 1 反对抗体。结论:导致 VCA 的 apoptotic 科罗拉多细胞死亡由于 caspases 的激活和 anti-apoptotic 蛋白质的抑制,部分地通过肿瘤坏死因素受体 1 个发信号小径。" @default.
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