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- W3148296098 abstract "导出色氨酸， indolic 代谢物在 Arabidopsis 天生的免疫拥有多样的功能到微生物引起的病原体感染。这里，我们在全身的获得的抵抗(SAR ) 由细菌的病原体 Pseudomonas syringae 触发了的 Arabidopsis 调查功能的角色和 indolic 新陈代谢的规章的特征。Indolic 新陈代谢广泛地在两 P 被激活。接种 syringae 、远，非接种的叶子。在接种地点， camalexin， indol-3-ylmethylamine (I3A ) ，和 indole-3-carboxylic，酸(集成通信适配器) 是主要积累混合物。Camalexin 累积被 MYB122，和细胞色素 P450 基因 CYP81F1 和 CYP81F2 断然影响。由对比，本地 I3A 生产由 PEN2- 依赖、独立的小径经由吲哚 glucosinolate 故障发生。而且，防卫荷尔蒙水杨酸酸的外长的申请在损坏它的先锋的情况下刺激 I3A 产生 indol-3-ylmethylglucosinolate (I3M ) ，和 SAR 管理者 pipecolic 酸为提高的 P 告知植物。indolic 新陈代谢的不同分支的导致 syringae 的激活。在 uninfected 全身的织物，新陈代谢的反应与 indolics I3A，集成通信适配器，和 indole-3-carbaldehyde (国际计算中心) 的提高的层次更特定、联系。全身的吲哚累积充分取决于功能的 CYP79B2/3， PEN2，和 MYB34/51/122，并且要求功能的 SAR 发信号。基因分析建议全身地提高的吲哚为 SAR 是非必需的并且联系水杨酸酸的全身的增加。然而，土壤成年然而并非水耕法地栽培的 cyp79b2/3 和 pen2 种，在 indolic 有缺点的两个第二等的新陈代谢，展出预先导致的免疫，它废除他们的内在的能力导致 SAR。" @default.
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- W3148296098 date "2016-01-01" @default.
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- W3148296098 title "Regulatory and Functional Aspects of Indolic Metabolism in Plant Systemic Acquired Resistance" @default.
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