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- W3149262013 abstract "An animal model of non hereditary AD was built by lesioning nucleus basalis of Meynert(nbM) and to investigate the behavioral alteration by Morris water maze, the pathological changes by special staining for senile plaques(SPs),enzymatic cytochemcal staining for AChE, and immunocytochemical staining for β amyloid protein (βAP) and τ protein. Transmission electron microscopic observation has also been made. Results showed: (1) loss of learning and memory ability; (2) occurrence of necrotic granules in cytoplasm and inclusion in axon hillock;accumulation of necrotic cell, and formation of SPs under light microscope; (3) accumulation of microtubules and formation of inclusion,increase of lysosome and edema and vacuolation of neurons and neuroterminals under electron microscopy; (4) decrease of synaptic density; (5) sharp decrease of AChE (acetylcholinesterase) positive fibers and neurons shown by immunocytochemical staining; (6)overexpression of βAP and τ protein." @default.
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- W3149262013 date "1997-01-01" @default.
- W3149262013 modified "2023-09-24" @default.
- W3149262013 title "An Animal Model of Non hereditary Alzheimers Disease and Its Behavioral and Pathologic Changes" @default.
- W3149262013 hasPublicationYear "1997" @default.
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