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- W3149308264 abstract "To the Editor: We appreciate the report by Takemoto et al. onTakotsubo cardiomyopathy without an identifiable cause in a patient undergoing long-term hemodialysis, which reflects the great diversity of cardiovascular events in end-stage renal disease patients. Although the etiology of Takotsubo cardiomyopathy remains unclear in most cases, we would like to bring to the readers’ attention an unusual cause of this stress cardiomyopathy. A 31-year-old woman with Chinese herb nephropathy and on continuous ambulatory peritoneal dialysis for 16 months developed severe chest pain rapidly after taking slimming pills containing triiodothyronine (128mg). Transient left ventricular apical ballooning without evidence of coronary artery abnormality was documented. In this patient, a burst of enhanced, excessive sympathetic activity arising from thyrotoxicosis factitia may have triggered a switch in intracellular signal trafficking in ventricular cardiomyocytes, from Gs protein to Gi protein signaling via the b2-adrenergic receptors. 2 This strategy counteracts the proapoptotic effect of excessive b1-adrenergic Gs protein pathway activation and seems to act as a physiological balance to prevent catecholamine-mediated myocardial damage. In our patient, the ventricular function and apical wall motion returned to normal 2 weeks later." @default.
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- W3149308264 date "2010-01-01" @default.
- W3149308264 modified "2023-09-23" @default.
- W3149308264 title "Antineutrophil cytoplasmic autoantibody-associated vasculitis on chronic dialysis" @default.
- W3149308264 hasPublicationYear "2010" @default.
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