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- W3149753896 abstract "不适当的 vascularization 限制骨头缺点的修理。以便改进 angiogenesis 并且加速成骨，有在骨头新生的基因修正的 co 有教养的房间的协同在这研究被调查。Endothelial 祖先房间(EPC ) 和骨头髓干细胞(BMSC ) 是有脉管的 endothelial 生长因素(VEGF ) 和骨头的基因的 transfected 形态基因的蛋白质 2 (BMP2 ) 由侵入人体气管粘膜的病菌分别地。co 有教养的房间，包括 BMSC + EPC， Ad-BMP2BMSC + EPC， BMSC + Ad-VEGFEPC，和 Ad-BMP2BMSC + 指定 Ad-VEGFEPC 组织为四个组，在 alginate 胶化上被播种然后肌内地植入进老鼠在 angiogenesis 和成骨上评估效果。VEGF 和 BMP2 能导致禁止者的 overexpression DNA 有约束力 1 (Id1 ) 显著地在 vitro 和增加支持了试管形成的基因在在培植以后的 Ad-BMP2BMSC + Ad-VEGFEPC 组的血容器的数量。不过， Id1 的 overexpression 延迟了造骨细胞和骨头形成的终端区别。后来，在 transcriptional 水平，钙小瘤，和碱的磷酸酶(高山) 活动的 osteogenic 基因表示证明渐渐的减少和最新形成的成骨区域的数量在 Ad-BMP2BMSC + Ad-VEGFEPC 组展出了小增加。这发现建议在 VEGFEPC 和 BMP2BMSCs 的 Id1 表示的一条平衡规定可能对为骨头新生的基于房间、基于基因的途径批评。" @default.
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- W3149753896 date "2011-01-01" @default.
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- W3149753896 title "BMP2 and VEGF promote angiogenesis but retard terminal differentiation of osteoblasts in bone regeneration by up-regulating Idl" @default.
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