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- W3149973681 abstract "瞄准：为了调查在肉吸入，香烟吸烟和 N-acetyltransferase 2 (NAT2 ) 之间的可能的协会， colorectal 癌症(CRC ) 上的基因多型性冒险。方法：有 CRC 的病人为性和年龄被匹配到健康控制。吸烟的肉吸入和香烟用一张特定的频率问询表被估计。DNA 从外部血被提取，多型性的遗传型被聚合酶链反应限制碎片长度多型性估计。五 NAT2 等位基因被学习(WT， M1， M2， M3 和 M4 ) 使用特定的消化酶。结果：有 colorectal 癌症(有结肠癌的 76 个女人和 90 个男人) 和 212 控制的 147 个病人的一个总数被学习。二个组的吝啬的年龄是 62 年。多于半这些题目(59.8% 在情况中组织， 51.9% 在控制组织) 是 NAT2 慢 acetylators。为 colorectal 癌症的机会比率是 1.38 (95% CI：0.90-2.12 ) 在慢 acetylators。尽管女人的数字是小的(76 在情况中组织的 n =) ，癌症风险被发现更低在里面中介(W/Mx ) acetylators [机会比率(或) ：0.55， 95% 信心间隔(95% CI ) ：0.29-1.02 ] 。这差别没在男人被观察(或：0.56， 95% CI：0.16-2.00 ) 。在 NAT2 快 acetylators (W/W 或 W/Mx ) 之中，超过 3 一个星期预定的肉消费增加了 colorectal 癌症的风险(或：2.05， 95% CI：1.01-4.16 ) 。相反，香烟吸烟在慢 acetylators 之中增加了 CRC 的风险(或：1.97， 95% CI：1.02-3.79 ) 。结论：CRC 的风险在快 acetylators 之中是更高的报导了更高的肉吸入。慢 NAT2 acetylation 与 CRC 的增加的风险被联系。" @default.
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- W3149973681 date "2011-01-01" @default.
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- W3149973681 title "N-Acetyltransferase 2 genetic polymorphisms and risk of colorectal cancer" @default.
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