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- W3150387277 abstract "RIG-I 是认出病毒的 RNA 的不同种类的一个枢轴的细胞质的传感器。这识别导致抄写因素 NF-B 和 IRF3，它协作导致打的 activationof 我干扰素(IFN ) 和天生的抗病毒的 response.In 这研究，我们作为 RIG-I-mediated IFN 的一个积极管理者识别了整齐的家庭蛋白质 TRIM4 感应。Overexpressionof TRIM4 加强了 IRF3 和 NF-B 的被触发病毒的激活，以及 IFN- 正式就职，而 TRIM4had 相反的效果击倒。机械学地， TRIM4 与 RIG-I 联系并且为连接 K63 的 polyubiquitination 指向它。我们 TRIM4 是由调停的导致病毒的 IFN 正式就职小径的一个重要管理者的 findingsdemonstrate 为 K63-linkedubiquitination 的 RIG-I。" @default.
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- W3150387277 date "2014-01-01" @default.
- W3150387277 modified "2023-09-23" @default.
- W3150387277 title "TRIM4 modulates type I interferon induction and cellular antiviral response by targeting RIG-I for K63-1inked ubiquitination" @default.
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