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- W3152302440 abstract "进在与危险性联系到 Crohn 的疾病的 NOD2 的三风险等位基因 R702W， G908R 和 1007fsInsC 的继承的调查例如越过种族和人口表明了异质的显著数量，与越过欧洲的地区性的变化，建议本地创始人效果。在非白种人人口， Crohn 的疾病继续在发生增加，但是这增加看起来不是在 NOD2 的变化的后果，进一步为另外的危险性部位推进积累的证据。已知的等位基因的频率处于健康和疾病越过人口被比较，为在 NOD2 的另外的等位基因的证据被考察。在染色体 16 上基于它的位置与一些另外的自体免疫的疾病危险性本地化重合，研究作为另外的自体免疫的混乱的潜在的原因指向了 NOD2 变化。当这些调查主要归还了否定调查结果，二疾病对主人疾病时，被显示了被风险等位基因在 NOD2 引起。这在复杂疾病调查是经常的，一些结果等候 NOD2 和差别的确认，而是鉴定在以内并且越过人口加薪关于在这个地点的变化的人口遗传的吸引人的问题。" @default.
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- W3152302440 date "2006-01-01" @default.
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- W3152302440 title "NOD2： Ethnic and geographic differences" @default.
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