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- W3153242912 endingPage "1912" @default.
- W3153242912 startingPage "1912" @default.
- W3153242912 abstract "Blood vessels in tumors are formed through a variety of different mechanisms, each generating vessels with peculiar structural, molecular, and functional properties. This heterogeneity has a major impact on tumor response or resistance to antineoplastic therapies and is now emerging as a promising target for strategies to prevent drug resistance and improve the distribution and efficacy of antineoplastic treatments. This review presents evidence of how different mechanisms of tumor vessel formation (vasculogenesis, glomeruloid proliferation, intussusceptive angiogenesis, vasculogenic mimicry, and vessel co-option) affect tumor responses to antiangiogenic and antineoplastic therapies, but also how therapies can promote alternative mechanisms of vessel formation, contributing to tumor recurrence, malignant progression, and acquired drug resistance. We discuss the possibility of tailoring treatment strategies to overcome vasculature-mediated drug resistance or to improve drug distribution and efficacy." @default.
- W3153242912 created "2021-04-26" @default.
- W3153242912 creator A5018599202 @default.
- W3153242912 creator A5059742725 @default.
- W3153242912 creator A5072467454 @default.
- W3153242912 date "2021-04-15" @default.
- W3153242912 modified "2023-10-14" @default.
- W3153242912 title "Alternative Vascularization Mechanisms in Tumor Resistance to Therapy" @default.
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