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- W3157484923 abstract "Epidemiological evidence establishes obesity as an independent risk factor for increased susceptibility and severity to viral respiratory pneumonias associated with H1N1 influenza and SARS-CoV-2 pandemics. Given the global obesity prevalence, a better understanding of the mechanisms behind obese susceptibility to infection is imperative. Altered immune cell metabolism and function are often perceived as a key causative factor of dysregulated inflammation. However, the contribution of adipocytes, the dominantly altered cell type in obesity with broad inflammatory properties, to infectious disease pathogenesis remains largely ignored. Thus, skewing of adipocyte-intrinsic cellular metabolism may lead to the development of pathogenic inflammatory adipocytes, which shape the overall immune responses by contributing to either premature immunosenescence, delayed hyperinflammation, or cytokine storm in infections. In this review, we discuss the underappreciated contribution of adipocyte cellular metabolism and adipocyte-produced mediators on immune system modulation and how such interplay may modify disease susceptibility and pathogenesis of influenza and SARS-CoV-2 infections in obese individuals." @default.
- W3157484923 created "2021-05-10" @default.
- W3157484923 creator A5006484997 @default.
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- W3157484923 creator A5040186552 @default.
- W3157484923 creator A5071611042 @default.
- W3157484923 date "2021-11-01" @default.
- W3157484923 modified "2023-10-14" @default.
- W3157484923 title "Adipocyte inflammation and pathogenesis of viral pneumonias: an overlooked contribution" @default.
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