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• W3161466561 abstract "This review highlights recent advances on the mechanisms and impact of HDL-small non-coding RNAs (sRNA) on intercellular communication in atherosclerosis.Studies demonstrate that HDL-microRNAs (miRNA) are significantly altered in atherosclerotic cardiovascular disease (ASCVD), and are responsive to diet, obesity, and diabetes. Immune cells, pancreatic beta cells, and neurons are shown to export miRNAs to HDL. In turn, HDL can deliver functional miRNAs to recipient hepatocytes and endothelial cells regulating adhesion molecule expression, cytokines, and angiogenesis. With high-throughput sRNA sequencing, we now appreciate the full sRNA signature on circulating HDL, including the transport of rRNA and tRNA-derived fragments. Strikingly, HDL were highly enriched with exogenous microbial sRNAs. HDL transport a diverse set of host and non-host sRNAs that are altered in cardiometabolic diseases. Given the bioactivity of these sRNAs, they likely contribute to cellular communication within atherosclerotic lesions, and are potential disease biomarkers and therapeutic targets." @default.
• W3161466561 created "2021-05-24" @default.
• W3161466561 creator A5025571380 @default.
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• W3161466561 date "2021-05-13" @default.
• W3161466561 modified "2023-10-03" @default.
• W3161466561 title "HDL-small RNA Export, Transport, and Functional Delivery in Atherosclerosis" @default.
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• W3161466561 doi "https://doi.org/10.1007/s11883-021-00930-7" @default.
• W3161466561 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/8455070" @default.
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• W3161466561 hasPublicationYear "2021" @default.
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