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- W3161470828 endingPage "147" @default.
- W3161470828 startingPage "136" @default.
- W3161470828 abstract "Cancer cachexia (CC) is a complex metabolic syndrome that accelerates muscle wasting and affects up to 80% of patients with cancer; however, timely diagnostic methods and effective cures are lacking. Although a considerable number of studies have focused on the mechanism of CC-induced muscle atrophy, few novel therapies have been applied in the last decade. In recent years, noncoding RNAs (ncRNAs) have attracted great attention as many differentially expressed ncRNAs in cancer cachectic muscles have been reported to participate in the inhibition of myogenesis and activation of proteolysis. In addition, extracellular vesicles (EVs), which function as ncRNA carriers in intercellular communication, are closely involved in changing ncRNA expression profiles in muscle and promoting the development of muscle wasting; thus, EV-related ncRNAs may represent potential therapeutic targets. This review comprehensively describes the process of ncRNA transmission through EVs and summarizes the pathways and targets of ncRNAs that lead to CC-induced muscle atrophy." @default.
- W3161470828 created "2021-05-24" @default.
- W3161470828 creator A5018690642 @default.
- W3161470828 creator A5019221988 @default.
- W3161470828 creator A5063588455 @default.
- W3161470828 creator A5064842058 @default.
- W3161470828 creator A5076988559 @default.
- W3161470828 date "2021-04-23" @default.
- W3161470828 modified "2023-09-27" @default.
- W3161470828 title "The mechanism by which noncoding RNAs regulate muscle wasting in cancer cachexia" @default.
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