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- W3167266846 endingPage "521" @default.
- W3167266846 startingPage "512" @default.
- W3167266846 abstract "Major Depressive Disorder is a major public health problem and has a high rate of treatment resistance. Fear conditioning has been proposed as a potential mechanism sustaining negative affect in mood disorders. With the aim of exploring cognitive effects of rapid-acting antidepressant treatments as a potential mechanism of action that can be targeted by neuromodulation, we performed a narrative review of the extant literature on effects of electroconvulsive therapy, ketamine or esketamine, and sleep deprivation on emotional/fear memory retrieval-reconsolidation. We explore interference with reconsolidation as a potential common pathway that explains in part the efficacy of rapid-acting antidepressant treatments with disparate mechanisms of action. We propose the testable hypothesis that fear learning circuits can be specifically targeted by neuromodulation to attempt rapid amelioration of depressive symptoms (especially repetitive negative thinking) while limiting unspecific, untoward cognitive side effects." @default.
- W3167266846 created "2021-06-22" @default.
- W3167266846 creator A5037178737 @default.
- W3167266846 creator A5052849134 @default.
- W3167266846 creator A5059339348 @default.
- W3167266846 date "2021-08-01" @default.
- W3167266846 modified "2023-10-07" @default.
- W3167266846 title "Cognitive effects of rapid-acting treatments for resistant depression: Just adverse, or contributing to clinical efficacy?" @default.
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